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RESEARCHERS REPORT FIRST SUCCESSFUL TREATMENT OF
CHRONIC TRAUMATIC BRAIN INJURY -- The treatment
involved a
new application and drug dosage of hyperbaric
oxygen therapy.

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atic+brain&op=ph
Story here...
http://www.earthtimes.
org/articles/show/news_press
_release,196429.shtml
Story below:
-------------------------
Researchers Report First Successful Treatment of
Chronic Traumatic Brain Injury
ARLINGTON, Va /PRNewswire/ -- The American Association of Health Freedom
and the International Hyperbaric Medical Association announced today that
physician-researchers at the Baromedical Research Institute and Louisiana
State University School of Medicine, New Orleans have reported the
successful treatment of chronic traumatic brain injury (TBI). The
findings, published in Brain Research (2007 Oct 12;1174:120-9), are
purportedly the first-ever demonstration of improvement of chronic brain
injury in animals. The treatment involved a new application and drug
dosage of hyperbaric oxygen therapy. Hyperbaric oxygen therapy has been
applied to diving injuries, carbon monoxide poisoning, "flesh-eating
bacteria," and chronic wounds. Application to chronic brain injury is
controversial. This demonstration is a landmark achievement that reverses
100 years of neurology and the institutionalized belief that chronic brain
injury in untreatable. It has significant implications for the treatment
of chronic human traumatic brain injury (TBI) such as for current U.S.
military veterans.
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Lead author, Paul G. Harch, M.D., Clinical
Assistant Professor, Director of the LSU School of Medicine Hyperbaric
Medicine Fellowship in New Orleans, and author of recently published The
Oxygen Revolution (Hatherleigh Press, NY), said that he made this
discovery when he first applied HBOT to divers with a disabling form of
"the bends," brain decompression illness. Harch noticed that the
traditional application of a single HBOT to freshly injured divers did not
result in the immediate cure described by the U.S. Navy. Instead, divers
from the Gulf of Mexico presented days to weeks after their injury and
required much more HBOT at a lower pressure. Eventually, he realized that
they were treating more chronic forms of brain injury since the bubbles
had long passed through the divers' brains. Harch and colleagues applied
this lower dose of HBOT to patients with other forms of chronic brain
injury, including trauma, cerebral palsy, autism, toxic brain injury,
dementia, and multiple other diagnoses.
Harch stated that when he presented this human case experience at
scientific meetings from 1992 to 1999 he was criticized for lack of an
animal model. As a result, he adapted the Feeney model of acute traumatic
brain injury to chronic brain injury and applied his human protocol of
HBOT. Harch and colleagues were able to show in Brain Research that they
could improve cognition (spatial learning and memory) while increasing
blood vessel density in the damaged hippocampus of the rats.
The potential application to chronic human TBI is important because there
is currently no treatment. Chronic TBI affects over 5 million Americans.
Costs of treating TBI exceed $60 billion/year in the U.S. alone. The
personal toll is far greater. Traumatic brain injury has also been
described as the signature injury of the wars in Iraq and Afghanistan. As
many as 400,000 U.S. servicemen have been exposed to blast injuries and
could have varying degrees of TBI. Application of HBOT to these veterans
could have a significant impact on their return to work, quality of life,
and national security. For information on the Brain Injury Rescue and
Rehabilitation Project, please visit
http://www.healthfreedom.net/.
-------------------------
Larry Scott --
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